Education

• B.S.  Kent State University  1984
• M.S.  John Carroll University  1989
• Ph.D.  Case Western Reserve University  1993
• PD  Cleveland Clinic Foundation  1996

Background

• 2017-present: Associate Professor with Tenure, Department of Environmental and Occupational Health, Indiana University, Bloomington, IN
• 2011-2016: Associate Professor, Department of Environmental and Occupational Health, Indiana University- Bloomington
• 2010-2011: Assistant Professor (visiting), Department of Environmental Health, Indiana University- Bloomington
• 2005-2010: Assistant Professor, Department of Pharmacology and Toxicology, Indiana University School of Medicine, Indianapolis, IN
• 2000-2004: Project Staff, Department of Cell Biology, Cleveland Clinic Foundation, Cleveland, OH
• 1996-2000: Research Associate, Department of Cell Biology, Cleveland Clinic Foundation, Cleveland, OH
• 1993-1996: Research Fellow, Department of Cell Biology, Cleveland Clinic Foundation, Cleveland, OH

Scholarly Interests

• Cancer Biology
• Pancreatic Cancer
• Chemical Carcinogenesis
• Toxicology of PFAS
• Obesity and Cancer

Selected Publications

Articles

Hocevar BA, Kamendulis LM, Pu X, Perkins SM, Wang ZY, Johnston EL, DeWitt JM, Li L, Loehrer PJ, Klaunig JE, Chiorean EG. (2014). Contribution of environment and genetics to pancreatic cancer susceptibility. PLoS One. 9(3):e90052

Kamendulis LM, Wu Q, Sandusky GE, Hocevar BA. (2014). Perfluorooctanoic acid exposure triggers oxidative stress in the mouse pancreas. Toxicol Rep. 1:513-521.

Chittiboyina S, Chen Z, Chiorean EG, Kamendulis LM, Hocevar BA. (2018) The role of the folate pathway in pancreatic cancer risk. PLoS One. 13(2):e0193298. doi: 10.1371/journal.pone.0193298. eCollection 2018. PMID: 29474406

Hocevar, BA (2019) Loss of Disabled-2 Expression in Pancreatic Cancer Progression.

Sci Rep. 9(1):7532. doi: 10.1038/s41598-019-43992-z. PMID: 31101868

Temkin, AM., Hocevar, BA., Andrews, DQ., Naidenko, OV., Kamendulis,LM.,(2020). Application of the Key Characteristics of Carcinogens to Per and Polyfluoroalkyl Substances (PFAS). Int. J. Environ. Res. Public. Health 17(5):1668. (PMID: 32143379)

Hocevar, SE., Kamendulis, LM., Hocevar, BA. (2020). Perfluorooctanic Acid (PFOA) activates the unfolded protein response in pancreatic acinar cells. J.Biochem. Mol. Toxicol. 34(11):e22561. doi:10.1002/jbt.22561. PMID: 32578922

Kamendulis, LM., Hocevar, JM., Sandusky, GE. & Hocevar, BA. (2022). Promotion of pancreatic cancer by perfluorooctanic acid (PFOA). Carcinogenesis 43(5):469-478. doi:10.1093/carcin/bgac005. (PMID: 35022659; PMCID: PMC9167031).